Autoimmunity complicating SARS-CoV-2 infection in selective IgA-deficiency.
Pfeuffer S; Pawlowski M; Joos GS; Minnerup J; Meuth SG; Dziewas R; Wiendl H
Forschungsartikel (Zeitschrift) | Peer reviewedZusammenfassung
Coronavirus disease 2019 (COVID-19) caused by SARS-CoV-2 can result in severe disease and become critically challenging to hospitals via high demand for intensive care and mechanical ventilation. Guillain-Barré syndrome (GBS) and its variants have been described as neurologic complications of COVID-19, and fatal cases were reported.1 The mechanisms by which COVID-19 predisposes to autoimmunity are unclear, and potential biomarkers or risk factors remain unknown.
Details zur Publikation
Fachzeitschrift: Neurology: Neuroimmunology and NeuroInflammation (Neurol Neuroimmunol Neuroinflamm)
Jahrgang / Bandnr. / Volume: 7
Ausgabe / Heftnr. / Issue: 6
Status: Veröffentlicht
Veröffentlichungsjahr: 2020 (28.11.2020)
Sprache, in der die Publikation verfasst ist: Englisch
Link zum Volltext: https://www.neurology.org/doi/10.1212/NXI.0000000000000881?url_ver=Z39.88-2003&rfr_id=ori:rid:crossref.org&rfr_dat=cr_pub%20%200pubmed
Stichwörter: Adult; Autoimmunity; Betacoronavirus; COVID-19; Coronavirus Infections; Female; Humans; IgA Deficiency; Pandemics; Plasma Exchange; Pneumonia, Viral; SARS-CoV-2
Autor*innen der Universität Münster
| Dziewas, Rainer | |
| Joos, Gunther | |
| Meuth, Sven | |
| Minnerup, Jens | |
| Pawlowski, Matthias | |
| Wiendl, Heinz Siegfried |