Embryonic cardiomyocytes can orchestrate various cell protective mechanisms to survive mitochondrial stress

Magarin M., Pohl T., Lill A., Schulz H., Blaschke F., Heuser A., Thierfelder L., Donath S., Drenckhahn J. (2016)

In: Journal of Molecular and Cellular Cardiology, 97(null).

Forschungsartikel (Zeitschrift)

Fine mapping of the 1p36 deletion syndrome identifies mutation of PRDM16 as a cause of cardiomyopathy

Arndt A., Schafer S., Drenckhahn J., Sabeh M., Plovie E., Caliebe A., Klopocki E., Musso G., Werdich A., Kalwa H., Heinig M., Padera R., Wassilew K., Bluhm J., Harnack C., Martitz J., Barton P., Greutmann M., Berger F., Hubner N., Siebert R., Kramer H., Cook S., Macrae C., Klaassen S. (2013)

In: American Journal of Human Genetics, 93(1).

Forschungsartikel (Zeitschrift)

Disruption of the murine dynein light chain gene Tcte3-3 results in asthenozoospermia

Rashid S., Grzmil P., Drenckhahn J., Meinhardt A., Adham I., Engel W., Neesen J. (2010)

In: Reproduction, 139(1).

Forschungsartikel (Zeitschrift)

3-D Imaging of Biomedical Samples

In: (Hrsg.), .

Compensatory Growth of Healthy Cardiac Cells in the Presence of Diseased Cells Restores Tissue Homeostasis during Heart Development

In: .

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Activation of Peroxisome Proliferator-Activated Receptor-δ as Novel Therapeutic Strategy to Prevent In-Stent Restenosis and Stent Thrombosis Hytönen J., Leppänen O., Braesen J., Schunck W., Mueller D., Jung F., Mrowietz C., Jastroch M., Von Bergwelt-Baildon M., Kappert K., Heuser A., Drenckhahn J., Pieske B., Thierfelder L., Ylä-Herttuala S., Blaschke F. (2016) In: Arteriosclerosis, Thrombosis, and Vascular Biology, 36(8). Forschungsartikel (Zeitschrift)
Embryonic cardiomyocytes can orchestrate various cell protective mechanisms to survive mitochondrial stress Magarin M., Pohl T., Lill A., Schulz H., Blaschke F., Heuser A., Thierfelder L., Donath S., Drenckhahn J. (2016) In: Journal of Molecular and Cellular Cardiology, 97(null). Forschungsartikel (Zeitschrift)
Impaired myocardial development resulting in neonatal cardiac hypoplasia alters postnatal growth and stress response in the heart Drenckhahn J., Strasen J., Heinecke K., Langner P., Yin K., Skole F., Hennig M., Spallek B., Fischer R., Blaschke F., Heuser A., Cox T., Black M., Thierfelder L. (2015) In: Cardiovascular Research, 106(1). Forschungsartikel (Zeitschrift)
Fine mapping of the 1p36 deletion syndrome identifies mutation of PRDM16 as a cause of cardiomyopathy Arndt A., Schafer S., Drenckhahn J., Sabeh M., Plovie E., Caliebe A., Klopocki E., Musso G., Werdich A., Kalwa H., Heinig M., Padera R., Wassilew K., Bluhm J., Harnack C., Martitz J., Barton P., Greutmann M., Berger F., Hubner N., Siebert R., Kramer H., Cook S., Macrae C., Klaassen S. (2013) In: American Journal of Human Genetics, 93(1). Forschungsartikel (Zeitschrift)
Heart development: Mitochondria in command of cardiomyocyte differentiation Drenckhahn J. (2011) In: Developmental Cell, 21(3). Forschungsartikel (Zeitschrift)
Disruption of the murine dynein light chain gene Tcte3-3 results in asthenozoospermia Rashid S., Grzmil P., Drenckhahn J., Meinhardt A., Adham I., Engel W., Neesen J. (2010) In: Reproduction, 139(1). Forschungsartikel (Zeitschrift)
Growth plasticity of the embryonic and fetal heart In: .
3-D Imaging of Biomedical Samples In: (Hrsg.), .
Mutations in sarcomere protein genes in left ventricular noncompaction In: .
Compensatory Growth of Healthy Cardiac Cells in the Presence of Diseased Cells Restores Tissue Homeostasis during Heart Development In: .